Doxorubicin induces senescence or apoptosis in rat neonatal cardiomyocytes by regulating the expression levels of the telomere binding factors 1 and 2. [electronic resource]
Producer: 20100217Description: H2169-81 p. digitalISSN:- 1522-1539
- Animals
- Animals, Newborn
- Anthracenes -- pharmacology
- Antibiotics, Antineoplastic -- toxicity
- Apoptosis -- drug effects
- Benzothiazoles -- pharmacology
- Cells, Cultured
- Cellular Senescence -- drug effects
- Checkpoint Kinase 2
- Cytoskeleton -- drug effects
- Dose-Response Relationship, Drug
- Doxorubicin -- toxicity
- Imidazoles -- pharmacology
- JNK Mitogen-Activated Protein Kinases -- antagonists & inhibitors
- Mitosis -- drug effects
- Myocytes, Cardiac -- drug effects
- Phenotype
- Protein Kinase Inhibitors -- pharmacology
- Protein Serine-Threonine Kinases -- metabolism
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Pyridines -- pharmacology
- RNA Interference
- Rats
- Rats, Sprague-Dawley
- Telomeric Repeat Binding Protein 1 -- genetics
- Telomeric Repeat Binding Protein 2 -- genetics
- Time Factors
- Toluene -- analogs & derivatives
- Tumor Suppressor Protein p53 -- antagonists & inhibitors
- bcl-2-Associated X Protein -- metabolism
- p38 Mitogen-Activated Protein Kinases -- antagonists & inhibitors
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Publication Type: Journal Article
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