Cholinergic receptor signaling modulates spontaneous firing of sinoatrial nodal cells via integrated effects on PKA-dependent Ca(2+) cycling and I(KACh). [electronic resource]
Producer: 20090921Description: H949-59 p. digitalISSN:- 1522-1539
- Action Potentials -- drug effects
- Animals
- Atropine -- pharmacology
- Bee Venoms -- pharmacology
- Calcium -- metabolism
- Calcium Channels, L-Type -- physiology
- Calcium Signaling -- drug effects
- Calcium-Binding Proteins -- metabolism
- Cells, Cultured
- Cesium -- pharmacology
- Chlorides -- pharmacology
- Cholinergic Agonists -- pharmacology
- Cyclic AMP -- metabolism
- Cyclic AMP-Dependent Protein Kinases -- metabolism
- Cyclic GMP -- metabolism
- Parasympatholytics -- pharmacology
- Patch-Clamp Techniques
- Pertussis Toxin -- pharmacology
- Phosphorylation -- drug effects
- Potassium Channel Blockers -- pharmacology
- Rabbits
- Receptors, Cholinergic -- physiology
- Sinoatrial Node -- cytology
- Stochastic Processes
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Publication Type: Journal Article; Research Support, N.I.H., Intramural
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