Activation of NF-kappaB by palmitate in endothelial cells: a key role for NADPH oxidase-derived superoxide in response to TLR4 activation. [electronic resource]
Producer: 20090908Description: 1370-5 p. digitalISSN:- 1524-4636
- Animals
- Aorta, Thoracic -- enzymology
- Bone Morphogenetic Protein 4 -- metabolism
- Cells, Cultured
- Dietary Fats -- administration & dosage
- Endothelial Cells -- drug effects
- Humans
- I-kappa B Proteins -- metabolism
- Intercellular Adhesion Molecule-1 -- metabolism
- Interleukin-1 Receptor-Associated Kinases -- metabolism
- Interleukin-6 -- metabolism
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myeloid Differentiation Factor 88 -- metabolism
- NADPH Oxidase 4
- NADPH Oxidases -- genetics
- NF-KappaB Inhibitor alpha
- NF-kappa B -- metabolism
- Onium Compounds -- pharmacology
- Palmitic Acid -- metabolism
- Phosphorylation
- RNA Interference
- RNA, Messenger -- metabolism
- Signal Transduction
- Superoxide Dismutase -- metabolism
- Superoxides -- metabolism
- Time Factors
- Toll-Like Receptor 4 -- deficiency
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural
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