Deletion of astrocyte connexins 43 and 30 leads to a dysmyelinating phenotype and hippocampal CA1 vacuolation. [electronic resource]
Producer: 20090717Description: 7743-52 p. digitalISSN:- 1529-2401
- Animals
- Animals, Newborn
- Astrocytes -- metabolism
- Basic Helix-Loop-Helix Transcription Factors -- metabolism
- Calcium-Binding Proteins
- Cell Proliferation
- Connexin 30
- Connexin 43 -- deficiency
- Connexins -- deficiency
- DNA-Binding Proteins -- metabolism
- Demyelinating Diseases -- complications
- Disease Models, Animal
- Gap Junctions -- pathology
- Glial Fibrillary Acidic Protein -- genetics
- Hippocampus -- pathology
- In Situ Nick-End Labeling -- methods
- Memory Disorders -- genetics
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Microfilament Proteins
- Microscopy, Electron -- methods
- Myelin Basic Protein -- metabolism
- Myelin Sheath -- pathology
- Nerve Tissue Proteins -- metabolism
- Oligodendrocyte Transcription Factor 2
- Phenotype
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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