Resolution of renal inflammation: a new role for NF-kappaB1 (p50) in inflammatory kidney diseases. [electronic resource]
Producer: 20090827Description: F429-39 p. digitalISSN:- 1522-1466
- Active Transport, Cell Nucleus
- Acute Disease
- Animals
- Antilymphocyte Serum
- Blotting, Southwestern
- Cells, Cultured
- Chemokines -- metabolism
- Disease Models, Animal
- Endothelial Cells -- metabolism
- Glomerulonephritis -- chemically induced
- Immunohistochemistry
- Kidney Glomerulus -- blood supply
- Lipopolysaccharides
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- NF-kappa B p50 Subunit -- deficiency
- NF-kappa B p52 Subunit -- metabolism
- Nephritis -- chemically induced
- Protein Multimerization
- Rats
- Rats, Wistar
- Remission, Spontaneous
- Time Factors
- Transcription Factor RelA -- metabolism
- Transcription Factor RelB -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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