Light responses in the mouse retina are prolonged upon targeted deletion of the HCN1 channel gene. [electronic resource]
Producer: 20090220Description: 2221-30 p. digitalISSN:- 1460-9568
- Amacrine Cells -- metabolism
- Animals
- Cyclic Nucleotide-Gated Cation Channels -- genetics
- Electroretinography
- Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels
- Immunohistochemistry
- Membrane Potentials -- genetics
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Neurons -- metabolism
- Organ Culture Techniques
- Patch-Clamp Techniques
- Photic Stimulation
- Photoreceptor Cells, Vertebrate -- metabolism
- Potassium Channels -- genetics
- Retina -- metabolism
- Retinal Bipolar Cells -- metabolism
- Retinal Ganglion Cells -- metabolism
- Synaptic Transmission -- genetics
- Vision, Ocular -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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