Azithromycin suppresses activation of nuclear factor-kappa B and synthesis of pro-inflammatory cytokines in tracheal aspirate cells from premature infants. [electronic resource]
Producer: 20071113Description: 483-8 p. digitalISSN:- 0031-3998
- Anti-Bacterial Agents -- pharmacology
- Azithromycin -- pharmacology
- Bronchopulmonary Dysplasia -- etiology
- Cell Count
- Cells, Cultured
- Dose-Response Relationship, Drug
- Female
- Gestational Age
- Humans
- I-kappa B Proteins -- metabolism
- Infant, Newborn
- Infant, Premature -- metabolism
- Interleukin-6 -- biosynthesis
- Interleukin-8 -- biosynthesis
- Male
- NF-KappaB Inhibitor alpha
- NF-kappa B -- metabolism
- Respiration, Artificial -- adverse effects
- Suction
- Trachea -- metabolism
- Tumor Necrosis Factor-alpha -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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