Hyperthermia activates a subset of ataxia-telangiectasia mutated effectors independent of DNA strand breaks and heat shock protein 70 status. [electronic resource]
Producer: 20070509Description: 3010-7 p. digitalISSN:- 0008-5472
- Animals
- Ataxia Telangiectasia Mutated Proteins
- Cell Cycle Proteins -- biosynthesis
- Cell Line
- DNA Damage
- DNA-Binding Proteins -- biosynthesis
- Embryo, Mammalian
- Fibroblasts -- metabolism
- HSP70 Heat-Shock Proteins -- biosynthesis
- Heat-Shock Response -- genetics
- Histones -- biosynthesis
- Humans
- Hyperthermia, Induced
- Mice
- Phosphorylation
- Protein Serine-Threonine Kinases -- biosynthesis
- Signal Transduction
- Tumor Suppressor Proteins -- biosynthesis
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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