Increase of carcinogenic risk via enhancement of cyclooxygenase-2 expression and hydroxyestradiol accumulation in human lung cells as a result of interaction between BaP and 17-beta estradiol. [electronic resource]
Producer: 20071004Description: 1606-12 p. digitalISSN:- 0143-3334
- Air Pollutants -- toxicity
- Animals
- Aryl Hydrocarbon Hydroxylases -- metabolism
- Benzo(a)pyrene -- toxicity
- Bronchi -- cytology
- Carcinogens, Environmental -- toxicity
- Cells, Cultured
- Cyclooxygenase 2 -- metabolism
- Cytochrome P-450 CYP1A1 -- metabolism
- Cytochrome P-450 CYP1B1
- Dinoprostone -- metabolism
- Drug Synergism
- Enzyme Activation
- Epithelial Cells -- metabolism
- Estradiol -- analogs & derivatives
- Estrogens, Catechol
- Fulvestrant
- Humans
- In Vitro Techniques
- Lung -- metabolism
- Male
- Membrane Proteins -- metabolism
- NF-kappa B -- metabolism
- Rats
- Rats, Sprague-Dawley
- Receptors, Estradiol -- antagonists & inhibitors
- Up-Regulation
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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