The lethality of Ku86 (XRCC5) loss-of-function mutations in human cells is independent of p53 (TP53). [electronic resource]
Producer: 20070215Description: 66-79 p. digitalISSN:- 0033-7587
- Cell Proliferation
- DNA Damage
- DNA Helicases -- biosynthesis
- DNA, Complementary -- metabolism
- DNA-Activated Protein Kinase -- metabolism
- Gene Expression Regulation
- Genetic Vectors
- Heterozygote
- Histones -- metabolism
- Humans
- Ku Autoantigen
- Models, Genetic
- Mutation
- Nuclear Proteins -- metabolism
- Radiation Tolerance
- Radiation, Ionizing
- Tumor Suppressor Protein p53 -- biosynthesis
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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