Arsenic trioxide induces apoptosis via the mitochondrial pathway by upregulating the expression of Bax and Bim in human B cells. [electronic resource]
Producer: 20070409Description: 313-8 p. digitalISSN:- 1019-6439
- Antineoplastic Agents -- pharmacology
- Apoptosis
- Apoptosis Inducing Factor -- metabolism
- Apoptosis Regulatory Proteins -- biosynthesis
- Arsenic Trioxide
- Arsenicals -- pharmacology
- B-Lymphocytes -- metabolism
- Bcl-2-Like Protein 11
- Cytochromes c -- metabolism
- Cytoplasm -- metabolism
- Humans
- Leukemia, Promyelocytic, Acute -- metabolism
- Male
- Membrane Potentials
- Membrane Proteins -- biosynthesis
- Mitochondria -- metabolism
- Oxidative Stress
- Oxides -- pharmacology
- Proto-Oncogene Proteins -- biosynthesis
- Up-Regulation
- bcl-2-Associated X Protein -- biosynthesis
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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