Tumor necrosis factor prevents alendronate-induced osteoclast apoptosis in vivo by stimulating Bcl-xL expression through Ets-2. [electronic resource]
Producer: 20051101Description: 2708-18 p. digitalISSN:- 0004-3591
- Alendronate -- pharmacology
- Animals
- Apoptosis
- Arthritis, Experimental -- drug therapy
- Bone Marrow Cells -- drug effects
- Cell Survival
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Immunoenzyme Techniques
- Mice
- Mice, Inbred C57BL
- Mice, Inbred CBA
- Mice, Transgenic
- Osteoclasts -- drug effects
- Proto-Oncogene Proteins -- genetics
- Proto-Oncogene Proteins c-bcl-2 -- genetics
- Proto-Oncogene Proteins c-ets
- RNA, Messenger -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Transcription Factors -- genetics
- Tumor Necrosis Factor-alpha -- genetics
- Up-Regulation
- bcl-X Protein
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
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