Sle1ab mediates the aberrant activation of STAT3 and Ras-ERK signaling pathways in B lymphocytes. [electronic resource]
Producer: 20050315Description: 1630-7 p. digitalISSN:- 0022-1767
- Animals
- Antibodies, Antinuclear -- biosynthesis
- B-Lymphocytes -- enzymology
- Cells, Cultured
- DNA-Binding Proteins -- metabolism
- Extracellular Signal-Regulated MAP Kinases -- metabolism
- Female
- Gene Expression Profiling
- Genetic Markers
- Genetic Predisposition to Disease
- Interleukin-6 -- physiology
- Lupus Erythematosus, Systemic -- enzymology
- MAP Kinase Signaling System -- genetics
- Mice
- Mice, Congenic
- Mice, Inbred C57BL
- Repressor Proteins -- biosynthesis
- STAT3 Transcription Factor
- Signal Transduction -- genetics
- Spleen -- immunology
- Suppressor of Cytokine Signaling 3 Protein
- Suppressor of Cytokine Signaling Proteins
- Trans-Activators -- metabolism
- Transcription Factors -- biosynthesis
- Up-Regulation -- genetics
- ras Proteins -- metabolism
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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