Deletion of murine Smn exon 7 directed to liver leads to severe defect of liver development associated with iron overload. [electronic resource]
Producer: 20041217Description: 1731-41 p. digitalISSN:- 0002-9440
- Animals
- Cyclic AMP Response Element-Binding Protein
- Exons
- Gene Deletion
- Genes, Dominant
- Heterozygote
- Homozygote
- Humans
- Immunoblotting
- Immunohistochemistry
- Integrases -- metabolism
- Iron -- metabolism
- Liver -- metabolism
- Mice
- Mutation
- Nerve Tissue Proteins -- genetics
- RNA Splicing
- RNA, Messenger -- metabolism
- RNA-Binding Proteins
- Reverse Transcriptase Polymerase Chain Reaction
- SMN Complex Proteins
- Survival of Motor Neuron 1 Protein
- Time Factors
- Transgenes
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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