Increased cardiac workload by closure of the ductus arteriosus leads to hypertrophy and apoptosis rather than to hyperplasia in the late fetal period. [electronic resource]
Producer: 20050201Description: 193-202 p. digitalISSN:- 0028-1298
- Animals
- Anti-Inflammatory Agents, Non-Steroidal -- toxicity
- Antimetabolites -- pharmacology
- Apoptosis
- Body Weight
- Bromodeoxyuridine -- pharmacology
- Cell Death
- Constriction, Pathologic -- chemically induced
- Ductus Arteriosus -- pathology
- Fetal Heart -- growth & development
- Gene Expression Regulation, Developmental
- Hyperplasia
- Hypertrophy -- etiology
- In Situ Hybridization
- In Situ Nick-End Labeling
- Indomethacin -- toxicity
- Organ Size
- Rats
- Rats, Wistar
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.