Nitric oxide attenuates cardiomyocytic apoptosis via diminished mitochondrial complex I up-regulation from cardiac ischemia-reperfusion injury under cardiopulmonary bypass. [electronic resource]
Producer: 20041026Description: 180-8 p. digitalISSN:- 0022-5223
- Animals
- Apoptosis -- drug effects
- Cardiopulmonary Bypass -- adverse effects
- Dogs
- Electron Transport Complex I -- drug effects
- Molsidomine -- analogs & derivatives
- Myocardial Reperfusion Injury -- enzymology
- Myocytes, Cardiac
- Nitric Oxide -- therapeutic use
- Nitric Oxide Donors -- therapeutic use
- Up-Regulation
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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