Molecular changes in normal appearing white matter in multiple sclerosis are characteristic of neuroprotective mechanisms against hypoxic insult. [electronic resource]
Producer: 20040112Description: 554-73 p. digitalISSN:- 1015-6305
- Aged
- Aged, 80 and over
- Antigens, CD -- metabolism
- Antigens, Differentiation, Myelomonocytic -- metabolism
- Aurora Kinases
- Blotting, Northern
- Brain -- metabolism
- CD3 Complex -- metabolism
- Case-Control Studies
- DNA-Binding Proteins -- metabolism
- Female
- Glial Fibrillary Acidic Protein -- metabolism
- Humans
- Hypoxia-Inducible Factor 1, alpha Subunit
- Immunohistochemistry
- In Situ Hybridization
- Male
- Middle Aged
- Models, Neurological
- Multiple Sclerosis -- genetics
- Myelin Basic Protein -- genetics
- Myelin Proteins
- Myelin-Associated Glycoprotein -- metabolism
- Myelin-Oligodendrocyte Glycoprotein
- Nuclear Proteins -- genetics
- Oligonucleotide Array Sequence Analysis -- methods
- Penicillamine -- analogs & derivatives
- Protein Serine-Threonine Kinases -- metabolism
- RNA, Messenger -- biosynthesis
- Reverse Transcriptase Polymerase Chain Reaction -- methods
- Transcription Factors -- metabolism
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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