Oncogenic Ras and Akt signaling contribute to glioblastoma formation by differential recruitment of existing mRNAs to polysomes. [electronic resource]
Producer: 20031210Description: 889-901 p. digitalISSN:- 1097-2765
- Animals
- Brain Neoplasms -- genetics
- Cell Cycle Proteins -- metabolism
- Cell Transformation, Neoplastic -- drug effects
- Cluster Analysis
- Enzyme Inhibitors -- pharmacology
- Gene Expression Regulation, Neoplastic -- genetics
- Glioblastoma -- genetics
- Humans
- Mice
- Mice, Transgenic
- Oligonucleotide Array Sequence Analysis
- Polyribosomes -- genetics
- Protein Biosynthesis -- drug effects
- Protein Serine-Threonine Kinases
- Proto-Oncogene Proteins -- antagonists & inhibitors
- Proto-Oncogene Proteins c-akt
- RNA, Messenger -- analysis
- Signal Transduction -- drug effects
- Tumor Cells, Cultured
- ras Proteins -- antagonists & inhibitors
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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