Rapid tumor development and potent vascularization are independent events in carcinoma producing FGF-1 or FGF-2. [electronic resource]
Producer: 20021223Description: 8128-39 p. digitalISSN:- 0950-9232
- Animals
- Autocrine Communication
- Carcinoma -- blood supply
- Disease Progression
- Endothelial Growth Factors -- metabolism
- Epithelial Cells -- pathology
- Female
- Fibroblast Growth Factor 1 -- genetics
- Fibroblast Growth Factor 2 -- genetics
- Gene Expression Regulation, Neoplastic
- Intercellular Signaling Peptides and Proteins -- metabolism
- Lymphokines -- metabolism
- Matrix Metalloproteinases -- biosynthesis
- Mesoderm
- Mice
- Mice, Nude
- Neoplasm Invasiveness
- Neoplasm Proteins -- physiology
- Neoplasm Transplantation
- Neovascularization, Pathologic -- genetics
- Phenotype
- Rats
- Receptor Protein-Tyrosine Kinases -- genetics
- Receptor, Fibroblast Growth Factor, Type 1
- Receptor, Fibroblast Growth Factor, Type 2
- Receptors, Cell Surface -- biosynthesis
- Receptors, Fibroblast Growth Factor -- genetics
- Receptors, Urokinase Plasminogen Activator
- Recombinant Fusion Proteins -- physiology
- Sequence Deletion
- Tissue Inhibitor of Metalloproteinase-2 -- biosynthesis
- Transfection
- Tumor Cells, Cultured -- metabolism
- Urinary Bladder Neoplasms -- blood supply
- Urokinase-Type Plasminogen Activator -- biosynthesis
- Vascular Endothelial Growth Factor A
- Vascular Endothelial Growth Factors
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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