beta-Amyloid neurotoxicity is exacerbated during glycolysis inhibition and mitochondrial impairment in the rat hippocampus in vivo and in isolated nerve terminals: implications for Alzheimer's disease. [electronic resource]
Producer: 20020816Description: 163-74 p. digitalISSN:- 0014-4886
- Adenosine Triphosphate -- metabolism
- Alzheimer Disease -- drug therapy
- Amyloid beta-Peptides -- administration & dosage
- Animals
- Disease Models, Animal
- Dizocilpine Maleate -- pharmacology
- Energy Metabolism -- drug effects
- Enzyme Inhibitors -- pharmacology
- Excitatory Amino Acid Antagonists -- pharmacology
- Glucose -- metabolism
- Glycolysis -- drug effects
- Hippocampus -- chemistry
- Injections, Intraperitoneal
- Iodoacetates -- pharmacology
- Male
- Microinjections
- Mitochondria -- chemistry
- Neocortex -- chemistry
- Neuroprotective Agents -- pharmacology
- Nitro Compounds
- Peptide Fragments -- administration & dosage
- Presynaptic Terminals -- chemistry
- Propionates -- pharmacology
- Pyruvic Acid -- pharmacology
- Rats
- Rats, Wistar
- Synaptosomes -- chemistry
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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