bFGF signaling and v-Myb cooperate in sustained growth of primitive erythroid progenitors. [electronic resource]
Producer: 20020214Description: 400-10 p. digitalISSN:- 0950-9232
- Adaptor Proteins, Signal Transducing
- Adaptor Proteins, Vesicular Transport
- Animals
- Blotting, Western
- Cell Differentiation -- drug effects
- Cell Division -- drug effects
- Cell Size -- drug effects
- Cells, Cultured
- Cellular Senescence -- drug effects
- Chick Embryo
- Erythrocytes -- cytology
- Erythroid Precursor Cells -- cytology
- Fibroblast Growth Factor 2 -- pharmacology
- Fibroblasts
- Flow Cytometry
- Gene Expression Regulation, Developmental -- drug effects
- Hemoglobins -- metabolism
- Isoenzymes -- metabolism
- Mitogens -- pharmacology
- Oncogene Proteins v-myb -- metabolism
- Phospholipase C gamma
- Proteins -- physiology
- Proto-Oncogene Proteins c-myb -- metabolism
- RNA, Messenger -- genetics
- Receptor Protein-Tyrosine Kinases -- metabolism
- Receptor, Fibroblast Growth Factor, Type 1
- Receptors, Fibroblast Growth Factor -- metabolism
- Shc Signaling Adaptor Proteins
- Signal Transduction -- drug effects
- Type C Phospholipases -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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