Grb3-3 is up-regulated in HIV-1-infected T-cells and can potentiate cell activation through NFATc. [electronic resource]
Producer: 20001113Description: 30925-33 p. digitalISSN:- 0021-9258
- Adaptor Proteins, Signal Transducing
- Adult
- Antibodies, Monoclonal -- metabolism
- Blotting, Western
- CD28 Antigens -- metabolism
- CD4-Positive T-Lymphocytes -- metabolism
- Cell Nucleus -- metabolism
- Cyclosporine -- pharmacology
- Cytoplasm -- metabolism
- DNA-Binding Proteins -- genetics
- Female
- GRB2 Adaptor Protein
- Gene Products, nef -- metabolism
- Gene Products, tat -- metabolism
- HIV Infections -- metabolism
- HIV-1 -- metabolism
- Humans
- Immunosuppressive Agents -- pharmacology
- Jurkat Cells
- Leukocytes, Mononuclear -- virology
- Luciferases -- metabolism
- MAP Kinase Kinase Kinase 1
- MAP Kinase Signaling System
- Male
- Middle Aged
- NFATC Transcription Factors
- Nuclear Proteins
- Plasmids -- metabolism
- Promoter Regions, Genetic
- Protein Isoforms
- Protein Serine-Threonine Kinases -- metabolism
- Proteins -- chemistry
- RNA, Messenger -- metabolism
- Receptors, Antigen, T-Cell -- metabolism
- Signal Transduction
- Terminal Repeat Sequences
- Time Factors
- Transcription Factor AP-1 -- metabolism
- Transcription Factors -- genetics
- Transfection
- Up-Regulation
- nef Gene Products, Human Immunodeficiency Virus
- tat Gene Products, Human Immunodeficiency Virus
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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