Alzheimer's beta-amyloid, human islet amylin, and prion protein fragment evoke intracellular free calcium elevations by a common mechanism in a hypothalamic GnRH neuronal cell line. [electronic resource]
Producer: 20000608Description: 14077-83 p. digitalISSN:- 0021-9258
- Alzheimer Disease -- metabolism
- Amyloid -- metabolism
- Amyloid beta-Peptides -- metabolism
- Calcium -- metabolism
- Cell Line, Transformed
- Cholesterol -- metabolism
- Gonadotropin-Releasing Hormone -- metabolism
- Homeostasis
- Humans
- Hypothalamus -- cytology
- Islet Amyloid Polypeptide
- Islets of Langerhans -- metabolism
- Neurons -- metabolism
- Prions -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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