A fusion protein of the estrogen receptor (ER) and nuclear receptor corepressor (NCoR) strongly inhibits estrogen-dependent responses in breast cancer cells. [electronic resource]
Producer: 20000110Description: 2122-36 p. digitalISSN:- 0888-8809
- Binding Sites
- Breast Neoplasms -- metabolism
- Cell Division
- Cell Line
- DNA -- metabolism
- Enzyme Inhibitors -- pharmacology
- Estradiol -- pharmacology
- Histone Acetyltransferases
- Histone Deacetylase Inhibitors
- Humans
- Hydroxamic Acids -- pharmacology
- Kidney
- Nuclear Proteins -- genetics
- Nuclear Receptor Co-Repressor 1
- Nuclear Receptor Coactivator 1
- Receptors, Estrogen -- genetics
- Recombinant Fusion Proteins -- pharmacology
- Repressor Proteins -- genetics
- Response Elements
- Transcription Factors -- metabolism
- Transcription, Genetic -- drug effects
- Tumor Cells, Cultured
No physical items for this record
Publication Type: Comparative Study; Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
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