Desensitization of cardiac beta-adrenoceptor signaling with heart failure produced by myocardial infarction in the rat. Evidence for the role of Gi but not Gs or phosphorylating proteins. [electronic resource]
Producer: 19990908Description: 1185-201 p. digitalISSN:- 0022-2828
- Adrenergic beta-Agonists -- pharmacology
- Animals
- Arrestins -- drug effects
- Autoradiography
- Blood Pressure
- Calcium -- pharmacology
- Catechols -- pharmacology
- Colforsin -- pharmacology
- Cyclic AMP-Dependent Protein Kinases -- drug effects
- G-Protein-Coupled Receptor Kinase 2
- GTP-Binding Protein alpha Subunits, Gi-Go -- drug effects
- GTP-Binding Protein alpha Subunits, Gs -- drug effects
- Gene Expression Regulation
- Heart Atria -- drug effects
- Heart Failure -- metabolism
- Heart Ventricles -- drug effects
- Isoproterenol -- pharmacology
- Myocardial Infarction -- metabolism
- Organ Size
- Pertussis Toxin
- Phosphorylation
- Propanolamines -- pharmacology
- RNA, Messenger
- Rats
- Receptors, Adrenergic, beta -- metabolism
- Signal Transduction
- Virulence Factors, Bordetella -- pharmacology
- beta-Adrenergic Receptor Kinases
- beta-Arrestins
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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